Home

Blind Fähigkeit verschwommen thapsigargin mechanism Simulieren Nach vorne Henne

IJMS | Free Full-Text | Valproate Attenuates Endoplasmic Reticulum  Stress-Induced Apoptosis in SH-SY5Y Cells via the AKT/GSK3β Signaling  Pathway
IJMS | Free Full-Text | Valproate Attenuates Endoplasmic Reticulum Stress-Induced Apoptosis in SH-SY5Y Cells via the AKT/GSK3β Signaling Pathway

SEC24A identified as an essential mediator of thapsigargin-induced cell  death in a genome-wide CRISPR/Cas9 screen | Cell Death Discovery
SEC24A identified as an essential mediator of thapsigargin-induced cell death in a genome-wide CRISPR/Cas9 screen | Cell Death Discovery

Molecules | Free Full-Text | Mipsagargin: The Beginning—Not the  End—of Thapsigargin Prodrug-Based Cancer Therapeutics
Molecules | Free Full-Text | Mipsagargin: The Beginning—Not the End—of Thapsigargin Prodrug-Based Cancer Therapeutics

Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A  Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction  | Chemical Research in Toxicology
Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction | Chemical Research in Toxicology

Endoplasmic reticulum stress stimulates the release of extracellular  vesicles carrying danger-associated molecular pattern (DAMP) molecules |  Oncotarget
Endoplasmic reticulum stress stimulates the release of extracellular vesicles carrying danger-associated molecular pattern (DAMP) molecules | Oncotarget

Proteomic Signatures in Thapsigargin-Treated Hepatoma Cells | Chemical  Research in Toxicology
Proteomic Signatures in Thapsigargin-Treated Hepatoma Cells | Chemical Research in Toxicology

Molecular mechanism of ER stress‐induced gene expression of tumor necrosis  factor‐related apoptosis‐inducing ligand (TRAIL) in macrophages - Huang -  2015 - The FEBS Journal - Wiley Online Library
Molecular mechanism of ER stress‐induced gene expression of tumor necrosis factor‐related apoptosis‐inducing ligand (TRAIL) in macrophages - Huang - 2015 - The FEBS Journal - Wiley Online Library

Knocking out the door to tunicamycin entry | PNAS
Knocking out the door to tunicamycin entry | PNAS

HRC promotes anoikis resistance and metastasis by suppressing endoplasmic  reticulum stress in hepatocellular carcinoma
HRC promotes anoikis resistance and metastasis by suppressing endoplasmic reticulum stress in hepatocellular carcinoma

Thapsigargin induces apoptosis when autophagy is inhibited in HepG2 cells  and both processes are regulated by ROS-dependent pathway - ScienceDirect
Thapsigargin induces apoptosis when autophagy is inhibited in HepG2 cells and both processes are regulated by ROS-dependent pathway - ScienceDirect

Cell death induced by the ER stressor thapsigargin involves death receptor  5, a non-autophagic function of MAP1LC3B, and distinct contributions from  unfolded protein response components | Cell Communication and Signaling |  Full Text
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text

SEC24A facilitates colocalization and Ca<sup>2</sup><sup>+</sup> flux  between the endoplasmic reticulum
SEC24A facilitates colocalization and Ca<sup>2</sup><sup>+</sup> flux between the endoplasmic reticulum

Thapsigargin promotes colorectal cancer cell migration through upregulation  of lncRNA MALAT1
Thapsigargin promotes colorectal cancer cell migration through upregulation of lncRNA MALAT1

MANF Ablation Causes Prolonged Activation of the UPR without  Neurodegeneration in the Mouse Midbrain Dopamine System | eNeuro
MANF Ablation Causes Prolonged Activation of the UPR without Neurodegeneration in the Mouse Midbrain Dopamine System | eNeuro

Molecules | Free Full-Text | Mipsagargin: The Beginning&mdash;Not the  End&mdash;of Thapsigargin Prodrug-Based Cancer Therapeutics
Molecules | Free Full-Text | Mipsagargin: The Beginning&mdash;Not the End&mdash;of Thapsigargin Prodrug-Based Cancer Therapeutics

Tunicamycin specifically aggravates ER stress and overcomes chemoresistance  in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation |  Journal of Experimental & Clinical Cancer Research | Full Text
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text

Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia
Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia

IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to  Anticancer Drug
IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to Anticancer Drug

Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in  Pharmacological Sciences
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences

Effect of endoplasmic reticulum calcium on paraquat‑induced apoptosis of  human lung type II alveolar epithelial A549 cells
Effect of endoplasmic reticulum calcium on paraquat‑induced apoptosis of human lung type II alveolar epithelial A549 cells

Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in  Pharmacological Sciences
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences

Cell death induced by the ER stressor thapsigargin involves death receptor  5, a non-autophagic function of MAP1LC3B, and distinct contributions from  unfolded protein response components | Cell Communication and Signaling |  Full Text
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text

Kinetics of Thapsigargin- Ca2+-ATPase (Sarcoplasmic Reticulum) Interaction  Reveals a Two-step Binding Mechanism and Picomolar Inhibition * - Journal  of Biological Chemistry
Kinetics of Thapsigargin- Ca2+-ATPase (Sarcoplasmic Reticulum) Interaction Reveals a Two-step Binding Mechanism and Picomolar Inhibition * - Journal of Biological Chemistry

Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA  Translation of the p53 Isoform p53/47 - ScienceDirect
Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA Translation of the p53 Isoform p53/47 - ScienceDirect